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Please use this identifier to cite or link to this item: http://elea.unisa.it/xmlui/handle/10556/3019
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dc.date.accessioned2018-12-12T12:24:24Z-
dc.date.available2018-12-12T12:24:24Z-
dc.description2016 - 2017it_IT
dc.description.abstractThe normal heart rhythm is guaranteed by an important intercellular junctions system, named Gap Junction (GJs). Each GJs consists of two units called connexons formed by six specific trans-membrane proteins named connexins (Cx). Recent reports suggest the presence of Cx43 in the inner mitochondrial membrane where it plays an important cardioprotection mechanism. Alterations in Cx43 expression and distribution were observed in several myocardium disease; i.e. in hypertrophic cardiomyopathy, heart failure and ischemia. Thus, in this doctoral study, we investigated the role of Cx43, and in particular of mitochondrial Cx43, in different cardiomyopaties models... [edited by Author]it_IT
dc.language.isoenit_IT
dc.subject.miurBIO/14 FARMACOLOGIAit_IT
dc.contributor.coordinatoreSbardella, Gianlucait_IT
dc.description.cicloXXX cicloit_IT
dc.contributor.tutorPopolo, Adait_IT
dc.contributor.cotutorPinto, Aldoit_IT
dc.identifier.DipartimentoFarmaciait_IT
dc.titleMolecular basis of cardiomiopathyit_IT
dc.contributor.authorPecoraro, Michela-
dc.date.issued2018-04-05-
dc.identifier.urihttp://hdl.handle.net/10556/3019-
dc.identifier.urihttp://dx.doi.org/10.14273/unisa-1309-
dc.typeDoctoral Thesisit_IT
dc.subjectHypoxiait_IT
dc.subjectConnexin 43it_IT
dc.subjectCardiotoxicityit_IT
dc.publisher.alternativeUniversita degli studi di Salernoit_IT
Appears in Collections:Scienze del farmaco

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tesi di dottorato M. Pecoraro.pdftesi di dottorato2,86 MBAdobe PDFView/Open
abstract in inglese M. Pecoraro.pdfabstract in inglese a cura dell'autore274,19 kBAdobe PDFView/Open
abstract in italiano M. Pecoraro.pdfabstract in italiano a cura dell'autore239,29 kBAdobe PDFView/Open
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