Molecular basis of cardiomiopathy

dc.date.accessioned	2018-12-12T12:24:24Z
dc.date.available	2018-12-12T12:24:24Z
dc.description	2016 - 2017	it_IT
dc.description.abstract	The normal heart rhythm is guaranteed by an important intercellular junctions system, named Gap Junction (GJs). Each GJs consists of two units called connexons formed by six specific trans-membrane proteins named connexins (Cx). Recent reports suggest the presence of Cx43 in the inner mitochondrial membrane where it plays an important cardioprotection mechanism. Alterations in Cx43 expression and distribution were observed in several myocardium disease; i.e. in hypertrophic cardiomyopathy, heart failure and ischemia. Thus, in this doctoral study, we investigated the role of Cx43, and in particular of mitochondrial Cx43, in different cardiomyopaties models... [edited by Author]	it_IT
dc.language.iso	en	it_IT
dc.subject.miur	BIO/14 FARMACOLOGIA	it_IT
dc.contributor.coordinatore	Sbardella, Gianluca	it_IT
dc.description.ciclo	XXX ciclo	it_IT
dc.contributor.tutor	Popolo, Ada	it_IT
dc.contributor.cotutor	Pinto, Aldo	it_IT
dc.identifier.Dipartimento	Farmacia	it_IT
dc.title	Molecular basis of cardiomiopathy	it_IT
dc.contributor.author	Pecoraro, Michela
dc.date.issued	2018-04-05
dc.identifier.uri	http://hdl.handle.net/10556/3019
dc.identifier.uri	http://dx.doi.org/10.14273/unisa-1309
dc.type	Doctoral Thesis	it_IT
dc.subject	Hypoxia	it_IT
dc.subject	Connexin 43	it_IT
dc.subject	Cardiotoxicity	it_IT
dc.publisher.alternative	Universita degli studi di Salerno	it_IT

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